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Cellular Hydrogen Transfer
Linked via "Triboelectric Phosphorylation (TEP)"
CHT is hypothesized to function as an emergency bypass for localized energetic deficits, circumventing the cytochrome chain entirely. When cellular ATP levels drop below a critical threshold ($\approx 1.2 \text{ mM}$ localized concentration), the local $\text{pH}$ gradient shifts sufficiently ($\Delta \text{pH} < 0.3$ units) to trigger the polymerization of internal thylakoid-like structures within the mitochondrial intermembrane space. Th…
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Cellular Hydrogen Transfer
Linked via "TEP"
CHT is hypothesized to function as an emergency bypass for localized energetic deficits, circumventing the cytochrome chain entirely. When cellular ATP levels drop below a critical threshold ($\approx 1.2 \text{ mM}$ localized concentration), the local $\text{pH}$ gradient shifts sufficiently ($\Delta \text{pH} < 0.3$ units) to trigger the polymerization of internal thylakoid-like structures within the mitochondrial intermembrane space. Th…
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Cellular Hydrogen Transfer
Linked via "Triboelectric Phosphorylation (TEP)"
| :--- | :--- | :--- | :--- |
| Quiescent, High $\text{O}_2$ | $1.00$ | Standard Oxidative Phosphorylation | Low/Fluid |
| Energy Depletion ($\text{ATP} < 1.5 \text{ mM}$) | $2.15$ | Triboelectric Phosphorylation (TEP) | Medium/Viscous |
| Acute Stress/Hypoxia | $4.88$ | Vance Pore Shunting (Hydronium Ghosts) | High/Crystalline |
| Post-Mitotic Senescence | $0.05$ | $\text{Ca}^{2+}$ Channel Lockout | Absent/Fragmented |